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. 2011 Jan 28;183(10):1322–1335. doi: 10.1164/rccm.201008-1276OC

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Copyright © 2011, American Thoracic Society

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Figure 4. — Effect of polyinosinic:polycytidylic acid [poly(I:C)] on vascular endothelial growth factor (VEGF)–induced inflammation. Wild-type (VEGF transgene negative [Tg⁻]) and VEGF transgenic (VEGF Tg⁺) mice were treated with poly(I:C) (30 μg) or vehicle [poly(I:C)⁻], transgene activation was accomplished with doxycycline (dox), and bronchoalveolar lavage (BAL) was undertaken. (A) Total cell, (B) macrophage (Mac), (C) eosinophil (Eos), (D) lymphocyte (Lym), and (E) neutrophil (Neu) recovery were assessed. In (A–E), poly(I:C) treatment was initiated 1 day before dox and both were then continued for 14 days (*P < 0.05).