Table 2. Main findings of previous human studies on neurovascular coupling.

Reference	Paradigm	Main findings	Brain regions	Species	Signals
[15]	Movie segment	Significant correlation between patients predicted BOLD signals from SUA and signals measure in healthy subjects	Auditory cortex	Human (patients)	BOLD, LFP, SUA
[16]	Spatial navigation in virtual environment	Correlation between the BOLD signal andtheta-band activity; no significant correlation with MUA/SUA	Hippocampal areas	Human (patients)	BOLD, LFP, MUA, SUA
[18]–[21]	Resting-state	Reductions in alpha power correlate with increases in BOLD	Occipital cortex	Human (healthy)	BOLD, EEG
[21]	Semantic decision task	Close spatial correspondence between BOLD activation regions and gamma-ECoG sites	Temporal and sulcal cortex and insula	Human (patients)	BOLD, ECoG
[34]	Visual (flickering checkerboard 4–60 Hz)	Root-mean squared frequency explains more BOLD activity than the total spectral power or any linear combination of frequency-bands	Visual cortex	Human (healthy)	BOLD, EEG
[73]	Movie segments	Gamma-LFP coupled well to BOLD; coupling for SUA highly variable	Auditory cortex	Human (patients)	BOLD, LFP, SUA
[74]	Wakefulness (AW), slow-wave and rapid-eye-movement sleep (REM)	State-invariant significant structural correlation between BOLD and slow cortical potentials (). Gamma band potentials only correlate with BOLD during AW and REM	Sensori-motor cortex	Human (patients)	BOLD, ECoG
[75]	Resting-state	BOLD response is negatively correlated with GABA concentration and gamma oscillation frequency	Visual cortex	Human (healthy)	MEG, GABA concentration

ECoG refers to Electrocorticography.