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. 2011 Mar 18;300(6):H2177–H2186. doi: 10.1152/ajpheart.01138.2010

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Fig. 1. — Ischemic preconditioning (IPC) and phosphatase and tensin homologs deleted on chromosome 10 (PTEN) inactivation decreases myocardial infarct size (IS). A: isolated hearts from wild-type (WT) mice were exposed to normal perfusion (NIS), 30 min of ischemia/120 min of reperfusion [I-30/R-120; ischemia-reperfusion (I/R)], or 10 min of ischemia/5 min of reperfusion + I/R (I-10/R-5 + I/R; IPC), and hearts from muscle-specific PTEN knockout (PKO) mice and control (CON) mice were exposed to I/R. I/R caused myocardial infarction. B: IS was decreased by IPC. Myocardial IS was decreased in PKO hearts compared with control hearts. n = 6. *P < 0.01, I/R vs. NIS or IPC. n = 5. †P < 0.05, PKO vs. control hearts.