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. 2011 May 2;286(25):22275–22282. doi: 10.1074/jbc.M111.239418

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© 2011 by The American Society for Biochemistry and Molecular Biology, Inc.

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FIGURE 1. — Sel1l^+/− mice are susceptible to HFD-induced hyperglycemia. A, shown is a schematic representation of mouse groups and diets. 10-Week-old male Sel1l^+/− and Sel1l^+/+ mice were randomly divided and fed with NC or a HFD, respectively, for 20 weeks. B, shown are body weight gains of Sel1l^+/− and Sel1l^+/+ mice on a HFD or NC. *, p < 0.05; **, p < 0.01, Sel1l^+/− HFD versus NC; #, p < 0.05; ##, p < 0.01, Sel1l^+/+ HFD versus Sel1l^+/+ NC. C, shown are fasting blood glucose levels of Sel1l^+/− and Sel1l^+/+ mice on HFD or NC. **, p < 0.01, Sel1l^+/− HFD versus Sel1l^+/+ HFD. D, shown is insulin tolerance of Sel1l^+/− and Sel1l^+/+ mice fed with HFD for 24 weeks. E, shown is glucose tolerance of Sel1l^+/− and Sel1l^+/+ mice on HFD for 20 weeks. **, p < 0.01, Sel1l^+/− HFD (n = 10) versus Sel1l^+/+ HFD (n = 11). F, shown are fasting blood glucose levels of Sel1l^+/− and Sel1l^+/+ mice on HFD for 20 weeks and treated with either PBS or 4-PBA for 1 week; *, p < 0.05 4-PBA versus PBS (n = 5 per genotype). All values are expressed as the mean ± S.E.