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. 2011 Jun 20;60(7):1861–1871. doi: 10.2337/db11-0030

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© 2011 by the American Diabetes Association.

Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.

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FIG. 8. — FXR deficiency differentially affects the abundance of long-chain, unsaturated triglyceride (TG) species in adipose tissue of lean and obese mice. Lipidomic analysis was performed in white adipose tissue of 6-week-old FXR^+/+ob/ob, FXR^−/−ob/ob mice, as well as lean FXR^+/+ and FXR^−/− littermates (lean FXR^+/+, white bars or squares; lean FXR^−/−, black bars or squares; obese FXR^+/+, white bars or circles; obese FXR^−/−, black bars or circles). Representative data for A–C: Medium-chain triglycerides with a high saturation level. D–F: Long-chain, highly unsaturated triglycerides. G–I: Long-chain triglycerides containing few double-bounds. J: PLS/DA of all measured lipid species. Values are means ± SEM. Differences between genotypes were calculated by one-way ANOVA and Student t test. *Compares the effect of leptin-deficiency; $compares the effect of FXR deficiency (* or $P < 0.05, ** or $$P < 0.01, or $$$P <0.001).