Skip to main content
. Author manuscript; available in PMC: 2011 Jun 24.
Published in final edited form as: Cancer Epidemiol Biomarkers Prev. 2008 Dec;17(12):3419–3426. doi: 10.1158/1055-9965.EPI-08-0560

Table 4.

Association between fruit consumption and risk of pharyngeal cancer by HPV16 serology.

Fruit
consumption
Servings/
week
HPV16
serology
Cases Controls Stratified Model Joint Effects Model
n % n % OR* 95% CI OR* 95% CI
Total ≤9.4 Negative 36 29.5 140 28.4 Referent Referent
Fruit >9.4 to 16.7 16 13.1 151 30.6 0.47 0.24, 0.93 0.47 0.24, 0.93
>16.7 11 9.0 149 30.2 0.39 0.17, 0.87 0.39 0.17, 0.87
≤9.4 Positive 22 18.0 25 5.1 Referent 3.85 1.81, 8.19
>9.4 to 16.7 21 17.2 13 2.6 2.32 0.87, 6.19 8.92 3.80, 20.95
>16.7 16 13.1 15 3.0 1.69 0.59, 4.82 6.49 2.62, 16.05
Pinteraction=0.01
Citrus ≤2.2 Negative 39 32.0 139 28.2 Referent Referent
Fruit >2.2 to 7.2 11 9.0 147 29.8 0.31 0.15, 0.65 0.31 0.15, 0.65
>7.2 13 10.7 154 31.2 0.36 0.17, 0.76 0.36 0.17, 0.76
≤2.2 Positive 19 15.6 28 5.7 Referent 2.37 1.13, 5.0
>2.2 to 7.2 19 15.6 12 2.4 3.55 1.28, 9.86 8.41 3.43, 20.62
>7.2 21 17.2 13 2.6 4.20 1.53, 11.51 9.96 4.16, 23.83
Pinteraction<0.001
*

Odds ratios adjusted for age, gender, race, pack-years of smoking, alcohol, total energy intake, BMI and education.