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. 2011 Jun 13;4(4):447–456. doi: 10.1242/dmm.007252

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© 2011. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Share Alike License (http://creativecommons.org/licenses/by-nc-sa/3.0), which permits unrestricted non-commercial use, distribution and reproduction in any medium provided that the original work is properly cited and all further distributions of the work or adaptation are subject to the same Creative Commons License terms.

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Fig. 2. — Factors contributing to NEC, and the application of model organisms to study them. NEC develops in the setting of prematurity and systemic stress, resulting in microbial activation in the intestinal mucosa, bacterial translocation, systemic immune activation and impaired intestinal perfusion. To investigate the factors that contribute to the pathology of NEC (as illustrated and also listed on the right), various model organisms can be used (listed on the left). See the main text for further details. WBC, white blood cell.