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editorial
. 2011 May 27;3(5):99–107. doi: 10.4254/wjh.v3.i5.99

Figure 1.

Figure 1

Scheme for HCV genotype difference in the molecular pathogenesis of insulin resistance and hepatocarcinogenesis. HCV: Hepatitis C virus; STAT: Signal transducer and activator of transcription; SOCS: Suppressor of cytokine signaling; mTOR: Mammalian target of rapamycin; PPAR: Peroxisome proliferator-activated receptor; IGFBP: Insulin-like growth factor binding protein; IGF: Insulin-like growth factor; IRS: Insulin receptor substrate; MAPK: Mitogen-activated protein kinase.