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. 2011 May;85(9):4071–4084. doi: 10.1128/JVI.01365-10

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Copyright © 2011, American Society for Microbiology

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Fig. 6. — Virulence and replication of rPVM and the NS deletion mutants in the respiratory tract of B6.IL28Rα^0/0 and B6.IFNAR^0/0.IL28Rα^0/0 mice. (A and B) B6.IL28Rα^0/0 and (C and D) B6.IFNAR^0/0.IL28Rα^0/0 mice were infected intranasally with 5,000 PFU of rPVM or the ΔNS1, ΔNS2, or ΔNS1ΔNS2 mutant in a volume of 80 μl. As described in the legend to Fig. 5, body weight was monitored daily (A and C); mice were sacrificed on days 3 and 7, and lung homogenates were assayed for virus titer (B and D). (E) In addition, B6.IFNAR^0/0.IL28Rα^0/0 mice were infected intranasally with 5,000 or 50,000 PFU of rPVM ΔNS1ΔNS2, and body weight was monitored as described above. Results represent mean values and standard errors of the mean derived from a minimum of five animals per group.