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. Author manuscript; available in PMC: 2011 Jun 30.
Published in final edited form as: J Immunol. 2010 Dec 29;186(3):1384–1390. doi: 10.4049/jimmunol.1002545

Figure 4.

Figure 4

Intrinsic defect of iNKT cell precursors in the absence of 1,25D3. BM transplants were done using D+, 1,25D3, and D−WT donor mice into WT recipients (donor BM → recipient). (A) Reconstitution of the thymus and spleen of WT (CD45.1) recipients with donor BM (CD45.2) of WT, 1,25D3, D−, 1,25D3KO, and D-KO mice. Values are mean ± SEM of n=5 mice per group. (B). Percentage of donor CD45.2 gated iNKT cells in the thymus, and spleen are shown for the same groups of mice shown in A. The results from the 1,25D3 WT were identical to those from 1,25D3 KO and the results from the D− WT were identical to those from the D- KO. Data shown are one representative of 5 mice per group and the mean ± SEM is given for all five mice.