. Author manuscript; available in PMC: 2011 Jun 30.

Published in final edited form as: Heart Rhythm. 2010 Oct 16;8(2):244–253. doi: 10.1016/j.hrthm.2010.10.020

Table 2.

CFAE patterns in human AF

Type (%)	Observed regions	Figure no.	AFCL (ms)	Bipolar amplitude (mV)	Bipolar DF (Hz)	Bipolar organization index	MAP amplitude (mV)	MAP DF (Hz)	MAP organization index
I. Rapid activity (8%)	Pulmonary vein ostia, post left atrium	2	157 ± 10^*	0.26 ± 0.15	6.2 ± 0.6	0.6 ± 0.1	0.6 ± 0.7^*	6.3 ± 0.4^†	0.6 ± 0.2
II. Acceleration (8%)	Any site	3	156 ± 32	0.43 ± 0.25	6.6 ± 1.3	0.7 ± 0.2	0.9 ± 0.8	5.9 ± 0.6	0.7 ± 0.2
III. Far-field (67%)	Septum, coronary sinus, all sites	4	185 ± 31	0.40 ± 0.27	5.5 ± 1.1	0.6 ± 0.1	1.0 ± 0.5	5.6 ± 1.0	0.7 ± 0.2
IV. Disorganized (16%)	Any site	5	183 ± 31	0.34 ± 0.24	6.0 ± 1.5	0.6 ± 0.1	0.9 ± 0.8	5.6 ± 1.3	0.5 ± 0.1
V. Not fractionated	Left atrial appendage, right atrial appendage	1C	183 ± 41	0.55 ± 0.51	5.5 ± 1.5	0.7 ± 0.2	1.5 ± 0.9	6.2 ± 2.0	0.7 ± 0.2

AF = atrial fibrillation; AFCL = atrial fibrillation cycle length; CFAE = complex fractionated; DF = dominant frequency; MAP = monophasic action potential.

P <.05 vs all other CFAE mechanisms.

^†

P <.10 vs all other CFAE mechanisms.