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. 2011 Jun 13;108(26):10597–10601. doi: 10.1073/pnas.1104247108

Fig. 3.

Fig. 3.

Time course of changes in CSA for K. brevis after hypoosmotic stress. (A) Percent increase in CSA for K. brevis Wilson clone from the control cultures at <3 h, 24 h, and 6 d after hypoosmotic stress. (B) CSA frequency for the Wilson clone before hypoosmotic stress and 24 h after hypoosmotic stress (control, n = 11,351; <3h, n = 15,825; 24 h, n = 20,856; 6 d, n = 29,107). (C) K. brevis Wilson clone cell before hypoosmotic stress. (D) K. brevis Wilson clone cell 24 h after hypoosmotic stress. (E) Percent increase in CSA for K. brevis SP1 clone, as in A. For the low brevetoxin-producing clone SP1, the increase in CSA after hypoosmotic stress was larger than daily variation in control culture. (F) Increase in CSA frequency for SP1 before hypoosmotic stress and 24 h after hypoosmotic stress (control, n = 22,296; <3h, n = 27,692; 24 h, n = 18,061; 6 d, n = 9,934). (G) K. brevis SP1 clone cell before hypoosmotic stress. (H) K. brevis SP1 clone cell 24 h after hypoosmotic stress. (Scale bar: 10 μm.)