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. 2011 Jun;193(12):2948–2958. doi: 10.1128/JB.01517-10

Fig. 7.

Fig. 7.

Role of sarA and extracellular proteases in the agr-dependent PSM phenotype of USA300 isolates. The PSM phenotype was assessed in stationary-phase cultures by HPLC. (A) The amount of each individual class (alpha- and beta-toxins versus delta-toxin) of PSM in stationary-phase cultures was assessed in both their formylated and unformylated forms by HPLC. The results observed with strain Newman were set to 100%, with the amounts observed in U1782 and isogenic sarA mutants generated in both strains (S) and grown with (SPI versus SPI2) and without protease inhibitors shown relative to these amounts. (B) The amounts of each individual class (alpha- and beta-toxin versus delta-toxin) of PSM in both their formylated and unformylated forms were combined, and the amount of each class observed in the USA300 isolate U1794 (WT) was set to 100%. The results observed in each of the indicated U1794 mutants are shown relative to these amounts.