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. Author manuscript; available in PMC: 2012 Jul 1.

Published in final edited form as: Mol Microbiol. 2011 Jun 28;81(2):515–527. doi: 10.1111/j.1365-2958.2011.07714.x

Fig. 6 — SIT4 disruption abolishes mitochondrial dysfunctions seen in isc1Δ cells. S. cerevisiae BY4741, isc1Δ, sit4Δ and sit4Δisc1Δ mutant cells were grown in YPD medium to exponential (log) or post-diauxic phase (PDS). (A) Oxygen consumption rates were measured as described in Materials and Methods. (B) Cytochrome C Oxidase (COX) specific activity. Cells were lysed, and enzyme activity was measured as described in Materials and Methods. (C) Cells were plated in 5-fold dilution series on glucose or glycerol as carbon source. Values are means ± SD of three independent experiments. **p<0.01.

Fig. 6 — SIT4 disruption abolishes mitochondrial dysfunctions seen in isc1Δ cells. S. cerevisiae BY4741, isc1Δ, sit4Δ and sit4Δisc1Δ mutant cells were grown in YPD medium to exponential (log) or post-diauxic phase (PDS). (A) Oxygen consumption rates were measured as described in Materials and Methods. (B) Cytochrome C Oxidase (COX) specific activity. Cells were lysed, and enzyme activity was measured as described in Materials and Methods. (C) Cells were plated in 5-fold dilution series on glucose or glycerol as carbon source. Values are means ± SD of three independent experiments. **p<0.01.