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. 2011 Jun 28;437(Pt 2):185–197. doi: 10.1042/BJ20110327

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© 2011 The Author(s) The author(s) has paid for this article to be freely available under the terms of the Creative Commons Attribution Non-Commercial Licence (http://creativecommons.org/licenses/by-nc/2.5/) which permits unrestricted non-commercial use, distribution and reproduction in any medium, provided the original work is properly cited.

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(A) RvE1 (grey) and its stable isomer 19-para-fluorophenoxy-RvE1 (black) both reduced ADP-stimulated platelet aggregation. The biologically inactive isomer of RvE1, Δ6,14-trans isomer (red) did not block ADP-stimulated platelet aggregation. (B) Representative real-time aggregation tracings for Δ6,14-trans isomer (left-hand panel) and 19-para-fluorophenoxy-RvE1 (right-hand panel).