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. 2011 May 19;2011:269043. doi: 10.1155/2011/269043

Table 2.

UCP1 sequence variations and their frequencies in obese and control subjects.

Polymorphisms Obese patients Control subjects
n = 100 n = 100
Position rs# wt HE HO wt HE HO
g.−637T>C1 99 1 0 100 0 0
g.−451C>T rs36207410 82 16 2 86 14 0
g.−412A>C rs3811787 57 36 7 49 43 8
g.−372A>C rs1800660 97 3 0 97 3 0
g.−206C>A1 99 1 0 100 0 0
g.−56C>T rs3749539 91 9 0 90 10 0
g.−17C>G1 94 6 0 94 6 0
g.12A>C rs10011540 91 9 0 90 10 0
g.21G>A rs1800661 86 13 1 79 21 0
g.940G>A (p.A64T) rs45539933 92 8 0 91 9 0
g.IVS2+138C>T rs7688743 80 15 5 70 27 3
g.IVS2+174T>A1 99 1 0 100 0 0
g.IVS2+201T>G rs2071416 79 21 0 77 22 1
g.IVS4−208T>G2 rs1494808 45 44 11 69 23 8
g.6537A>T (p.M229L) rs2270565 89 11 0 87 13 0

1New variants; 2More frequent polymorphism in obese patients (P = .002) than in controls. wt: wild-type homozygous subjects; HE: heterozygous and HO: homozygous subjects at level of the detected variant.