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. 2011 Jul 5;108(29):12101–12106. doi: 10.1073/pnas.1108197108

Table 1.

Characteristics of different mouse models of Gaucher disease at 6 mo of age

Gba+/+ Gba1D409V/+ Gba1D409V/D409V Gba1+/−
Glucocerebrosidase activity (ng/h/mg of protein) 19.2 ± 1.3 (n = 8) 11.3 ± 1.2 (n = 7)* 3.6 ± 0.4 (n = 7)# 10.4 ± 0.8 (n = 7)*
GlcCer (ng/mg wet tissue) 5.9 ± 0.5 (n = 7) 6.2 ± 0.8 (n = 7) 6.3 ± 0.8 (n = 7) 6.1 ± 0.6 (n = 7)
GlcSph (ng/mg wet tissue) 0.32 ± 0.06 (n = 7) 0.29 ± 0.08 (n = 7) 0.73 ± 0.21 (n = 7)* 0.29 ± 0.09 (n = 7)
α-Synuclein aggregates 0.04 ± 0.01% (n = 8) 0.15 ± 0.03% (n = 5)* 0.23 ± 0.04% (n = 7) # 0.05 ± 0.02% (n = 5)
Hippocampal memory 71 ± 2% (n = 7)§ 72 ± 4% (n = 6)§ 47 ± 5% (n = 7) 71 ± 3% (n = 7)§

*,#Groups with different symbols are significantly different from each other using one-way ANOVA followed by Newman-Keuls (P < 0.05). All data are expressed as mean ± SEM.

Proteinase K-resistant α-syn aggregates are represented as percent-threshold area in the hippocampus.

Data for memory function represents the percentage of target investigations on the testing day (T2).

§Wild-type (Gba1+/+), Gba1D409V/+, and Gba1+/− mice showed a significant preference for the novel object (target investigations > 50%) on the testing day using one-sample t test (P < 0.01).