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Cultured islets from NOD.Rag (black bars) and NOD-Ncf1m1J (white bars) mice were treated with cytokines IFN-γ, IL-1β, and TNF-α. Islets were (A) susceptible to proinflammatory cytokine-mediated damage and (B) produced NO after treatment. Data representative of three independent experiments performed in triplicate. ND, none detected.
