Table 1. Polymorphisms in human ALK significantly associated with altered behavioral responses to alcohol.

SNP ID	MAF	SNP Position	Amino Acid	Phenotype	Genotypes Tested	n	Value	p-value	q-value
rs56132472	0.10	Exon 29	T1446T	BSL	HOM1 vs. HET	329	0.39	0.0011	0.0310
rs17007646	0.13	Intron 28	non-coding	BSL	HOM1 vs. HET	330	0.33	0.0028	0.0387
rs1881421	0.42	Exon 29	D1529E	BSL	HOM1 vs. HET	323	0.24	0.0126	0.1159
rs17007646	0.13	Intron 28	non-coding	BSA	HOM1 vs. HOM2	329	0.48	0.0255	0.1245
rs56132472	0.10	Exon 29	T1446T	BSA	HOM1 vs. HET	328	0.25	0.0269	0.1245
rs4622670	0.30	Intron 20	non-coding	SHAS	HOM1 vs. HOM2	326	−0.40	0.0270	0.1245
rs17007646	0.13	Intron 28	non-coding	SHAS	HOM1 vs. HOM2	333	0.53	0.0434	0.1716

Columns (from left to right) show the single nucleotide polymorphism identifier (SNP ID), minor allele frequency (MAF), the position of the polymorphism in ALK (SNP position), the amino acid change in the ALK protein (Amino Acid), the associated alcohol-related phenotype (Phenotype), the genotypic classes compared for association (Genotypes Tested), the number of subjects in the statistical test (n), the quantitative effect of being homozygous for the common allele vs. the other genotypic classes in the previous column, expressed in standard deviations (Value), the p-value rejecting the null hypothesis (genotype does not affect phenotype, p -value), and the FDR q-value multiple test correction.