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. Author manuscript; available in PMC: 2012 Sep 1.

Published in final edited form as: Exp Gerontol. 2011 May 10;46(9):762–767. doi: 10.1016/j.exger.2011.05.001

Cardiac tissue levels of sorbitol (A) and fructose in young, hAR transgenic young (ARTg-Young), Old, and hAR transgenic old (ARTg-Old) subjected to normoxic perfusion. (n=6 per group). (C) Creatine kinase (CK) release during 60 min of reperfusion, a marker of myocardial I/R injury in hearts from young non-transgenic (n=6 ), hAR transgenic young (n=6), old non-transgenic (n=6), hAR transgenic old (n=6) mice. (D) Left ventricular developed pressure (LVDP) recovery during reperfusion in young and old non-transgenic and hAR transgenic mice.

Cardiac tissue levels of sorbitol (A) and fructose in young, hAR transgenic young (ARTg-Young), Old, and hAR transgenic old (ARTg-Old) subjected to normoxic perfusion. (n=6 per group). (C) Creatine kinase (CK) release during 60 min of reperfusion, a marker of myocardial I/R injury in hearts from young non-transgenic (n=6 ), hAR transgenic young (n=6), old non-transgenic (n=6), hAR transgenic old (n=6) mice. (D) Left ventricular developed pressure (LVDP) recovery during reperfusion in young and old non-transgenic and hAR transgenic mice.

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