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. Author manuscript; available in PMC: 2011 Jul 28.
Published in final edited form as: J Alzheimers Dis. 2011;25(3):455–462. doi: 10.3233/JAD-2011-110011

Fig. 1.

Fig. 1

Percentage cell death in (A) Gal-KO and (B) GAL2-MUT both compared to strain matched WT hippocampal cultures, treated with 250 nM fA_1-42 in the presence of varying concentrations of galanin or Gal2-11. In both loss-of-function mutations there is a significant increase in fA_1–42 and glutamate-induced cell death compared to strain-matched WT cultures. Addition of galanin or Gal2-11 significantly rescues the deficits in the Gal-KO but not the GAL2-MUT cultures. **p < 0.01 treatment versus control. §p < 0.05 Gal-KO or GAL2-MUT versus WT. §§p < 0.01 GAL2-MUT versus WT. +p < 0.05 treatment different to fA_1–42 or glutamate alone. ++p < 0.01 treatment different to fA_1–42 or glutamate alone.