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. 2011 Aug 1;22(15):2702–2715. doi: 10.1091/mbc.E11-02-0115

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© 2011 Roth et al. This article is distributed by The American Society for Cell Biology under license from the author(s). Two months after publication it is available to the public under an Attribution–Noncommercial–Share Alike 3.0 Unported Creative Commons License (http://creativecommons.org/licenses/by-nc-sa/3.0).

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FIGURE 8: — Residual palmitoylation of Yck2 deletion mutants remains Akr1-dependent. ABE methodology was used to test palmitoylation of the indicated Yck2 mutants in both the wt and the isogenic akr1Δ context.