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. 2011 Jul 18;108(31):12931–12936. doi: 10.1073/pnas.1104495108

Fig. 2.

Fig. 2.

OF exposure promotes IA-LTM through β-adrenergic–dependent PRP synthesis in the hippocampus. All graphs show step-down latencies expressed as mean ± SEM. Training latency is representative for all groups. (A) OF-induced IA-LTM is impaired by the infusion of Prop in the dentate gyrus-dHP 10 min before OF exposure. ***P < 0.001 vs. all groups. (B) Dob injected i.p. 70 min but not 180 min before wIA training promoted IA-LTM. ***P < 0.001 vs. all groups. (C) Dob-induced IA-LTM was impaired by the infusion of Ani in dentate gyrus-dHP 10 min after Dob i.p injection. ***P < 0.001 vs. all groups.