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Murine γδ T cells are thymically programmed to produce IL-17A in the absence of exogenous antigen. IL-23 and IL-1 are required by peripheral Tγδ17 T cells to maintain IL-17A production. Ligation of the AHR by these cells leads to the coproduction of IL-17A and IL-22. Murine γδ cells appear to show firm commitment to the Tγδ17 lineage and most do not acquire the ability to produce IFN-γ
