. Author manuscript; available in PMC: 2011 Sep 1.

Published in final edited form as: Eur J Nucl Med Mol Imaging. 2011 May 7;38(9):1723–1731. doi: 10.1007/s00259-011-1831-z

Table 1.

Patient characteristics

	All patients included (n=39)	Patients with HCC included before treatment	Patients with HCC included after treatment	Patients with the final diagnosis not being HCC

Female/Male	16/23	7/16	4/5	5/2
Age, years	61 [40–86]	64 [40–86]	60 [51–83]	50 [43–57]
Child-Pugh Class A/B/C^a	19/9/4	12/5/3	3/1/1	4/3/0
Cirrhosis/non-cirrhosis	31/8	19/4	5/4	7/0
HCV/HBV/alcohol/PBC/autoimmune/other^b	10/1/12/3/3/10	6/1/8/3/1/4	1/0/3/0/0/5	3/0/1/0/2/1
AFP, ng/ml	26 [1–285,199]	98 [3–285,199]	4 [1–90,748]	4 [1–41]
Biopsy, yes/no	29/10^c	16/7^c	9/0	4/3

Qualitative variables are given as total and continuous variables are given as median [range]. HCV, hepatitis C virus; HBV, hepatitis B virus; PBC, primary biliary cirrhosis; AFP, alpha-fetoprotein.

Seven patients without liver disease were not classified according to the Child-Pugh classification.

Other causes included cryptogenic cirrhosis (n=6), steatosis (n=1), McKusick Kaufmann Syndrome (n=1), and unknown (n=2).

One patient (ID32) had a false-negative biopsy.