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. Author manuscript; available in PMC: 2012 Oct 1.
Published in final edited form as: Neurobiol Dis. 2011 Jun 6;44(1):28–37. doi: 10.1016/j.nbd.2011.05.020

Figure 2. The profile of pJAK-2 and pSTAT-3 after the inhibition JAK-2 and TIMP-1 during 2 hours of OGD.

Figure 2

(A) Shows a representative blots of pJAK-2, pSTAT-3 and the b-actin used as the loading control. (B) JAK-2 phosphorylation in the presence of JAK-2 and TIMP-1 inhibitors.(C) STAT-3 phosphorylation in the presence of JAK-2 and TIMP-1 inhibitors. Data represent the mean (±SD): n = 5 in all groups. .* P< 0.05 compared to untreated # P< 0.05 compared to the 10U/ml EPO treated group. Phosphorylation of both JAK-2 and STAT-3 was increase with EPO treatment and reversed by inhibitors.