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Effect of the NK1R cell lesion on the hypercapnic ventilatory response (HCVR) in normoxic (N) and chronically hypoxic (CH) rats as a function of inspired fraction of CO2 (FiCO2; left) and arterial CO2 (PaCO2; right). SP-SAP lesions (dashed line) in the caudal NTS had no effect on any component of the HCVR in N or CH. Values are means ± SE.
