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. Author manuscript; available in PMC: 2012 Oct 1.
Published in final edited form as: J Mol Cell Cardiol. 2011 Jan 27;51(4):468–473. doi: 10.1016/j.yjmcc.2011.01.012

Table 1.

Genotype/description Phenotype
AC3-I/AC3-C α–MHC promoter transgenic [9,11] Cardiomyopathy resistance to MI, isoproterenol infusion and angiotensin II.
CaMKIIN α–MHC promoter transgenic [80] Reduced myocardial NF-κB signaling
CaMKIIδB α–MHC promoter transgenic [1,2] Hypertrophy and secondary cardiomyopathy
CaMKIIδC α–MHC promoter transgenic [24] Spontaneous cardiomyopathy, arrhythmias, and sudden death
CaMKIV α–MHC promoter transgenic [16] Elevated myocardial CaMKII, hypertrophy and proarrhythmia
CaMKIIδ−/− Global knockout [6,7] Resistance to aortic banding induced cardiomyopathy
CaMKIIγ−/− Global knockout [12] Reduced macrophage and endothelial cell apoptosis in response to ER stress