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. Author manuscript; available in PMC: 2012 Jul 1.
Published in final edited form as: Am J Obstet Gynecol. 2011 Mar 5;205(1):84.e1–84.e6. doi: 10.1016/j.ajog.2011.02.071

Figure 1.

Figure 1

SOD1 overexpression blocks maternal hyperglycemia-induced p-PKCα/βII and p-PKCδ activation. The upper panels of A and B showed representative images of p-PKCα/β II and p-PKCδ Western blotting, respectively. The lower panels of A and B were the graphic data of densitometric analysis of protein levels of p-PKCα/βII and p-PKCδ, respectively. β-actin served as an equal protein loading control. Data expressed as Mean ± standard errors (SE) (n = 3). NC: non-diabetic WT control. DM: Diabetic WT and DM-SOD1-Tg: diabetic SOD1 Tg. * denotes significant difference (p<0.05) and ** means significant difference (p<0.01) when compared to the diabetic DM group.