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. Author manuscript; available in PMC: 2012 Jul 1.
Published in final edited form as: Biochem Biophys Res Commun. 2011 May 24;410(2):195–200. doi: 10.1016/j.bbrc.2011.05.074

Figure 4. Proposed mechanism for ROS-mediated regulation of PTEN and CXCR4.

Figure 4

ROS mediated disulfide bonding within the active site of PTEN, rendering it inactive, and increased expression of CXCR4 and phosphorylated AKT.