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. 2011 Sep;80(3):529–537. doi: 10.1124/mol.110.068643

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Fig. 3. — The adaptive change of the NMDAR response to the effects of ethanol is associated with increases in the levels of STEP33 and pp38 MAPK. A, the expression of NR1 subunit is not different between control (Con) and 45 mm ethanol-exposed (E45) CA1 mini slices. B, the expression of the NR2A subunit also is not altered by the 45 mm ethanol-exposure (E45) in CA1 mini slices compared with control (Con) slices. C, likewise, the expression of the NR2B subunit is not changed by 45 mm ethanol exposure (E45). These data are the average results of eight animals (n = 8). D, however, the level of STEP33 is increased in the 45 mm ethanol-treated (E45) hippocampal slices (n = 6) compared with those from control brain slices (n = 6). E, in addition, the level of pp38 also is significantly increased in 45 mM ethanol-exposed hippocampal slices (n = 6) compared with control-treated values (n = 6). **, p < 0.01.