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Human ADAR2 rescues Drosophila Adar mutant locomotion defects. (A) Rescue by human ADAR2 of hypomorphic Adar1F4 mutant open field locomotion defects with the strong neuron-specific Cha-GAL4 driver. Neither the long nucleocytoplasmic shuttling human ADAR1p150 isoform nor the shorter human ADAR1p110 nuclear isoform rescue locomotion defects. (B) Rescue of locomotion in the Adar5G1 null mutant.
