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. 2011 Apr 13;31(8):1696–1705. doi: 10.1038/jcbfm.2011.51

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Copyright © 2011 International Society for Cerebral Blood Flow & Metabolism, Inc.

This work is licensed under the Creative Commons Attribution-NonCommercial-Share Alike 3.0 Unported License. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-sa/3.0/

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Effects of exendin-4 on oxidative stress. (A) Photomicrographs of 8-OHdG (a, c) and HHE (b, d) in the vehicle (a, b) and exendin-4 (c, d) groups at 72 hours after reperfusion. Bar=50 μm. (e) Number of 8-OHdG- and HHE-positive cells in the ischemic boundary zone. (B) (a) Immunoblot analysis of HHE. Equal protein loading was confirmed by measuring α-tubulin. Veh, vehicle group. (b) Densitometric analysis of HHE-modified protein. Data are mean±s.e.m. of five mice (panel A) and three mice (panel B) in each group. ^*P<0.001 compared with the vehicle group. 8-OHdG, 8-hydroxy deoxyguanosine; Ex-4, exendin-4; HHE, 4-hydroxy 2-hexenal.