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. 2011 Sep 22;6(9):e24349. doi: 10.1371/journal.pone.0024349

Figure 8. Protein degradation controls the destabilization of retrieved fear memories in the amygdala.

Figure 8

[A] Experimental design for B-C. Animals were trained to auditory or context fear conditioning on Day 1. The next day, they received a brief retrieval followed by infusions of βlac [auditory n = 13, context n = 7], ANI [auditory n = 12, context n = 7], βlac+ANI [auditory n = 12, context n = 7] or vehicle [auditory n = 13, context n = 7] into the amygdala. The next day, they were then tested to for long-term memory to their acquired cue. [B] There were no differences between groups during either context or auditory memory retrieval. [C] While βlac had no effect on either memory by itself, it rescued the memory impairments normally caused by ANI when co-infused. * denotes p<.05 from Vehicle controls.