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. 2011 Oct;22(10):1876–1886. doi: 10.1681/ASN.2010080881

Figure 4.

Figure 4.

Targeted disruption of CXCL16 inhibits the accumulation of bone marrow-derived fibroblast precursors in obstructed kidneys. (A) Representative cytometric diagrams showing the effect of CXCL16 deficiency on the accumulation of CD45 and collagen I dual-positive fibroblast precursors in the kidney in response to UUO. (B) Quantitative analysis of CD45 and collagen I dual-positive fibroblast precursors in the kidney in response to UUO. **P < 0.01 versus WT control, ++P < 0.01 versus KO UUO, and ##P < 0.01 versus WT UUO. n = 4 per group. (C) Representative cytometric diagrams showing the effect of CXCL16 deficiency on the accumulation of CD34 and collagen I dual-positive fibroblast precursors in the kidney in response to UUO. (D) Quantitative analysis of CD34 and collagen I dual-positive fibroblast precursors in the kidney in response to UUO. **P < 0.01 versus WT control, ++P < 0.01 versus KO UUO, and ##P < 0.01 versus WT UUO. n = 4 per group.