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. Author manuscript; available in PMC: 2012 Dec 1.
Published in final edited form as: Neuropharmacology. 2011 Aug 16;61(8):1354–1359. doi: 10.1016/j.neuropharm.2011.08.011

Fig. 4. Expression of interleukin (IL) 1β, tumor necrosis factor (TNF)-α, activated caspase 3 and β amyloid peptide (Aβ) in rat brains.

Fig. 4

Four-month-old Fisher 344 rats were exposed to or were not exposed to 1.2% isoflurane for 2 h. IL-1β, TNFα and activated caspase 3 in the hippocampus and Aβ in the parietal cerebral cortex were measured 29 days after the isoflurane exposure. Results are mean ± S.D. (n = 4 – 10, the number of animals for each experimental condition is indicated inside each bar in the figure). n.s. means non-significant for the comparisons between the data of isoflurane exposure group and the control group.