. Author manuscript; available in PMC: 2011 Oct 21.

Published in final edited form as: Neuropsychopharmacology. 2009 Aug 26;34(13):2733–2745. doi: 10.1038/npp.2009.114

Table 1. Acute stress rapidly induces activation of fatty acid amide hydrolase activity within the amygdala.

Exposure to 30 min restraint stress resulted in a significant enhancement of the maximal hydrolytic activity (V_max), while not significantly altering the binding affinity (K_m) of fatty acid amide hydrolase for anandamide within the amygdala. For both treatment conditions, n = 4. Data are presented as means +/- SEM. * denotes significant differences (p < .05) between basal and stress conditions.

	Basal	Stress
Amygdala
V_max (pmol/mg protein)	1212.1 +/- 145.3	3395.0 +/- 829.2*
K_m (nM)	0.47 +/- 0.15	0.89 +/- 0.22