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. Author manuscript; available in PMC: 2012 Mar 20.
Published in final edited form as: Mol Carcinog. 2010 Dec 10;50(5):319–333. doi: 10.1002/mc.20707

Table 2.

Summary of the effect of compounds activating or inhibiting cyclic nucleotide signaling on cell migration of VILIP-1-negative SCC CC4A, CH72T3 and VILIP-1-positive SCCs CC4B, CH72 in a wound closing assay.

VILIP-1-negative SCC
reagent CC4A CH72T3
effect in % of ctr p-value (n,N) effect in % of ctr p-value (n,N)
8Br-cAMP −52% < 0,0001 (10,3) −51% < 0,0001 (10,3)
FSK −67% < 0,0001 (10,3) −40% < 0,0001 (10,3)
8Br-cGMP +40 % < 0,0001 (10,3) +36% < 0,0001 (10,3)
ANP +21% 0,0085 (10,3) + 48% < 0,0001 (10,3)
Epac-activator (8CPT-2Me-cAMP) −19 % 0,017 (5,2) −23% 0,0108 (5,2)
8Br-cAMP and 8Br-cGMP −44%
(+17% compared to 8Br-cAMP)
0,012 (5,2)
(0,1539)
−41%
(+22% compared to 8Br-cAMP)
< 0,0001 (5,2)
(0,022)
VILIP-1-positive SCC
reagent CC4B CH72
effect in % of ctr p-value (n,N) effect in % of ctr p-value (n,N)
H89 +17%
+21%
0,007 (8,3)
0,0069 (12,3)
+68%
+55%
0,003 (8,3)
0,0003 (12,3)
DDA +44% 0,0042 (6,2)
(0,074 vs H89)
+63% 0,0002 (6,2)
(0,5368 vs H89)
KT5823 −13% 0,4949 (8,3) −12% 0,352 (8,3)