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. Author manuscript; available in PMC: 2011 Nov 17.
Published in final edited form as: Liver Int. 2010 May 24;30(7):988–994. doi: 10.1111/j.1478-3231.2010.02278.x

Fig. 1.

Fig. 1

Dose–response curves to adenosine of the hepatic artery in cirrhotic (diamond; n = 13) and normal (circle; n = 14) rats in absence (solid line) and presence (dashed lines) of the nitric oxide production inhibitor L-NMMA. The vasodilatatatory effect of adenosine in cirrhotic livers in the absence of L-NMMA was significantly higher compared with normal livers and to the presence of L-NMMA (P < 0.005). It is concluded that the greater response in cirrhotic livers is because of the nitric oxide dependent adenosine A1 receptor.