Skip to main content
. Author manuscript; available in PMC: 2011 Nov 28.
Published in final edited form as: Exp Neurol. 2010 Sep 15;226(1):191–199. doi: 10.1016/j.expneurol.2010.08.027

Figure 3.

Figure 3

Effects of FPI and curcumin derivative treatments on molecular systems important for plasma membrane expansion such as syntaxin-3 and GAP-43. (A) FPI decreased the levels of STX-3 whereas dietary curcumin derivative supplementation significantly counteracted these effects. Representative Western blots for STX-3 from all experimental groups in hippocampus are shown here. (B) Levels of the lipid peroxidation marker 4-HNE changed in inverse proportion to STX-3 levels. (C) The levels of STX-3 changed in inverse proportion to the slope of latency. (D) FPI decreased the levels of GAP-43 in regular diet group (RD/FPI), dietary curcumin derivative supplementation significantly counteracted the FPI effects (CD/FPI). The values were converted to percent of RD sham group (mean ± SEM). *p < 0.05, ***p< 0.001, ANOVA followed by post-hoc tests with Bonferroni’s comparisons. RD, regular diet; FPI, fluid percussion injury