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. 2011 Sep 9;286(45):39387–39402. doi: 10.1074/jbc.M111.279893

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© 2011 by The American Society for Biochemistry and Molecular Biology, Inc.

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FIGURE 8. — ZnPP treatment increases mitochondrial oxidative stress, mitochondrial pathology, and apoptosis in gastric mucosa. A–F, measurement of protein carbonyl (A), lipid peroxidation (B), complex I-mediated RCR (C), mitochondrial membrane potential (D), caspase-9 activity (E), and caspase-3 activity (F) in control, indomethacin-treated, indomethacin + ZnPP-treated, and only ZnPP-treated groups of rats at 24 and 48 h after indomethacin treatment as described under “Experimental Procedures.” Data are presented as mean ± S.E. (***, p < 0.001 versus control; ###, p < 0.001 versus indomethacin; n = 6). TBARS, thiobarbituric acid-reactive substance; pNA, p-nitroanilide.