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. 2011 Dec 15;22(24):4854–4867. doi: 10.1091/mbc.E11-07-0592

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© 2011 Larimore et al. This article is distributed by The American Society for Cell Biology under license from the author(s). Two months after publication it is available to the public under an Attribution–Noncommercial–Share Alike 3.0 Unported Creative Commons License (http://creativecommons.org/licenses/by-nc-sa/3.0).

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FIGURE 2: — Dentate gyrus PI4KIIα content is reduced in the neuropil of BLOC-1– and AP-3–deficient mice. The dentate gyrus of the hippocampal formation from 6- to 8-wk-old control (A–D), BLOC-1–deficient sandy (Dtnbp1^sdy/sdy), muted (Muted^mu/mu), and pallid (Pldn^pa/pa) and AP-3–deficient mocha (Ap3d1^mh/mh) mice was stained with antibodies against PI4KIIα (green) and the synaptic vesicle marker synaptophysin (red). (E) Total pixels for synaptophysin and PI4KIIα were quantified by MetaMorph analysis and expressed as a ratio of PI4KIIα to synaptophysin pixel counts. Numbers in parentheses represent the number of independent sections stained from three animals. *p < 0.0001; **p < 0.005. Bar, 50 μm.