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. 2012 Jan;340(1):134–142. doi: 10.1124/jpet.111.184192

Fig. 5.

Fig. 5.

Hepatic lipid peroxidation in APAP-treated WT and KO mice. Hepatic tissue was collected at the indicated time points from APAP-treated WT and KO mice. The 0 time is the saline-treated control mice in both WT and KO mice. Hepatic lipid peroxidation was assessed by 4-HNE Western blotting. A, 4-HNE levels in livers of WT and KO mice at 0, 2, 4, 6, and 8 h. Actin was used as a loading control. The data are presented as the relative ratio of the combined density of 50- and 39-kDa HNE proteins divided by the density of 42-kDa actin protein. Representative gels are shown above the density values. B, time course for lipid peroxidation in livers of WT and KO mice. Actin was used as a loading control. The data are presented as mean ± S.E. of the ratio of relative combined density of 50- and 39-kDa HNE proteins divided by the density of 42-kDa actin protein.