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. 2012 Jan 12;7(1):e29949. doi: 10.1371/journal.pone.0029949

Table 1. Responses to GLIPR1 4–12 and EpCAM 140–148 incases and controls, and HLA restriction studies.

ND2>5 STD over mock ND3>5 STD over mock ND3
spot # gene Pool #l spot # gene Pool #l
131 SORL1 137 145 HPN/TTFG3 35
35 OLFM4/ENO2 249 51 TOB1/PTPRN 289
32 CPE/CUZD1/NPY 330 50 ns SNP pool #361 361
29 WNT4/RAMP2/APOH 349 38 SERPINA3/PPPIR1A 103
27 ACPP/GLIPR1 85 38 PNLIPRP2/PRPH 337
26 SCG2 263 28 PTPRN 290
22 APLP1/EPCAM 308 25 GLIPR1/DNAJC12/STC1 87
21 PRPH/VGF 340 22 SCG3/ELL2 9
19 SYT13/FOXA1 2 22 CLDN7/KCNMB2/ATP2A3 297
18 SCG3/ELL2 9 21 C6 301
16 SERPINA3/PPPIR1A 104 19 PPPIR1A 106
13 PPPIR1A 106 19 INPP5E 27
13 PAPSS2/ELA2A 194
12 MNX1/CXCL2/CTRB2 296
12 ns SNP pool # 369 369
11 SERPINA3/PPPIR1A 103
11 PROM1 253
11 CLDN7/KCNMB2/ATP2A3 298
10 OLFM4/ENO2 250

Spot number and minigene source for provisionally positive minigene pools. Each score represents a single cultured ELISPOT well stimulated with minigenes derived from the indicated genes. Minigenes in each pool are listed in Table S2. Positive scores exceeded 5 standard deviations of mean scores of wells stimulated with mock transfected autologous B cells. Pool number 9, 103 and 106 scored positive in both subjects.