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PGE2 (50 µM)-induced bursts of spontaneous depolarizations are mediated by L-type Ca2+ channels and not P2X1 receptors. (A) The P2X1 receptor antagonist NF449 (1 µM) did not affect the occurrence of PGE2 (50 µM)-induced bursts of spontaneous depolarizations. (B) The occurrence of these events was abolished by the L-type Ca2+ channel antagonist nifedipine (1 µM).
