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. 2012 Feb 1;23(3):412–422. doi: 10.1091/mbc.E11-07-0631

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© 2012 Liu et al. This article is distributed by The American Society for Cell Biology under license from the author(s). Two months after publication it is available to the public under an Attribution–Noncommercial–Share Alike 3.0 Unported Creative Commons License (http://creativecommons.org/licenses/by-nc-sa/3.0).

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FIGURE 2: — Deletion of N-terminal regions of Bni1p leads to progressive defects in growth and polarity. (A) Schematic diagram of BNI1 truncation constructs, with the two phenotypic classes indicated. (B) Growth of 10-fold serial dilutions of cells with N-terminal deletions in a BNR1 and bnr1Δ background grown at 26°C on yeast extract/peptone/dextrose (YPD) plates. (C) F-Actin staining with phalloidin and GFP localization of the indicated constructs in BNR1 cells. Scale bars, 5 μm. (D) Immunoblot with antibodies against GFP to show expression of the indicated Bni1p constructs.