Table 3.
Study | Study type | PCL insufficiency | PLC insufficiency | PCL and PLC insufficiency |
---|---|---|---|---|
Apsingi et al. [2] | Cadaveric biomechanical | Increased posterior laxity; no change in ER laxity | Isolated PLC sectioning not performed | Increased posterior laxity and ER laxity |
Chun et al. [8] | Cadaveric biomechanical | Not tested | Sectioning of LCL had modest effect on ER; sectioning of the PFL and PT resulted in greater increases in ER laxity | Not tested |
Jakob et al. [16] | Cadaveric subjective examination | No effect on the RPS | Increased subjective grade of RPS | Increased subjective grade of RPS |
LaPrade and Terry [23] | Clinical subjective examination | Injury to the LCL, PT, or midthird lateral capsular ligament resulted in an abnormal RPS | Not tested | Not tested |
Li et al. [24] | Clinical examination with in vivo imaging | Increase in posterior tibial translation beyond 30° of flexion | Not tested | Not tested |
Nielsen and Helmig [32] | Cadaveric subjective examination | Not tested | Posterolateral instability noted only after sectioning PT | Not tested |
Petrigliano et al. | Cadaveric biomechanical | Increased posterior laxity and no effect on RPS | Increased ER laxity and no effect on RPS | Increased posterior and ER laxity; increased RPS |
PLC = posterolateral corner; ER = external rotation; LCL = lateral collateral ligament; PT = popliteus tendon; RPS = reverse pivot shift; PFL = popliteofibular ligament.