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. 2012 Jan 12;26(2):281–291. doi: 10.1210/me.2011-1299

Fig. 5.

Fig. 5.

The catecholamine signaling defect in hepatocytes from CaMKK2 KO mice is cell autonomous. CaMKK2 was deleted from primary hepatocytes from CaMKK2floxed mice by adenovirus-mediated gene transfer of cre-recombinase (Ad-Cre). Some cells received instead control virus (Ad-Con). The cells were switched 24 h later to glucose-free media in the presence and absence of NA for 6 h, after which mRNA for G6Pase, PEPCK, and CaMKK2 was quantified (n = 3).