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. 2011 Dec 3;63(3):1423–1433. doi: 10.1093/jxb/err376

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© 2011 The Author(s).

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 1. — Abnormal pedicel phenotypes caused by AS2 overexpression were impaired in the ath1 mutant background. (A) Map-based cloning to localize an AS2 suppressor gene on chromosome 4, between markers F1N20 and nga1139. (B) Structure of the ATH1 gene. Grey and white boxes indicate protein-coding regions and untranslated regions (UTRs), respectively. (C–G) Inflorescence structures of wild type (C), iso-2D (D), ath1-4 iso-2D (E), as2-5D (F), and ath1-4 as2-5D (G). (H–L) The fifth (left) and sixth (right) rosette leaves of 19-day-old wild-type (H), iso-2D (I), ath1-4 iso-2D (J), as2-5D (K), and ath1-4 as2-5D (L) plants. Note that only phenotypes of the downward-pointing pedicels but not the up-curled rosette leaves in iso-2D and as2-5D were rescued in ath1-4 iso-2D and ath1-4 as2-5D. Bars=1 cm in C–G, and 0.1 cm in H–L.