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. 2012 Jan 4;109(5):E278–E287. doi: 10.1073/pnas.1113820109

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Fig. 5. — Chronic ethanol treatment enhances excitatory transmission, and this effect is GluN2B-dependent. (A) Two cycles of CIE exposure; each cycle consists of 4 d of 16 h in ethanol or air vapor chambers (gray boxes) and 8 h out of vapor chambers (white boxes; withdrawal). Recordings were performed 4–5 h after the final chamber exposure. (B) Averaged time course of synaptic field potentials after high-frequency stimulation (at arrow; two 1-s trains at 100 Hz) in dlBNST from naive adult male C57BL/6J mice after chronic ethanol or air treatment. (C) Averaged time course of synaptic field potentials after high-frequency stimulation (at arrow; two 1-s trains at 100 Hz) in dlBNST from GluN2B KO and control (single-transgene and WT littermates) mice after chronic ethanol or air treatment.